lu C, Katar S, Doru O, Ta MA. Henna-induced hemolytic anemia and acute renal failure. Turk J Pediatr 2001;43:65–6.

11 Dawson JP, Thayer WW, Desforges JF. Acute hemolytic anemia in the newborn infant due to naphthalene poisoning. Report of two cases with investigation into the mechanism of the disease. Blood 1958;13:1113–25.

12 Schafer WB. Acute hemolytic anemia related to naphthalene. Report of a case in a newborn infant. Pediatrics 1951;7:172–4.

13 Siegel E, Wason S. Mothball toxicity. Pediatr Clin North Am 1986;33:369–74.

14 Roth B, Bulla M, von Lilien T, Statz A, Okonek S. Clinical findings and treatment of paraquat poisoning in childhood. Monatsschr Kinderheilk 1983;131:458–63.

15 Weinbaum Z, Samuels SJ, Schenker MB. Risk factors for occupational illnesses associated with the use of paraquat (1,1′-dimethyl-4,4′-bipyridylium dichloride) in California. Arch Environ Health 1995;50:341–8.

16 André N, Paut O, Arditti J et al. Intoxication grave au dichromate de potassium après introduction nasale accidentelle. Arch Pèdiatr (Paris) 1998;5:145–8.

Cutaneous Symptoms Following Ingestion, Inhalation and Injection of Some Toxicants

Systemic poisoning by ingestion, inhalation or injection of some toxicants (see Box 184.2) may produce cutaneous symptoms. However, the reports in infants are very few.

Dioxins

After an accident in a chemical plant in Seveso, Italy, 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) spread over a populated area. The entire population, including children, was affected and contamination took place not only through direct exposure but also through inhalation and ingestion of contaminated foods, especially fruits and vegetables.

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